Apraxia in movement disorders
Identifieur interne : 002A13 ( Main/Exploration ); précédent : 002A12; suivant : 002A14Apraxia in movement disorders
Auteurs : Cindy Zadikoff [Canada] ; Anthony E. Lang [Canada]Source :
- [ 0006-8950 ]
Descripteurs français
- Pascal (Inist)
English descriptors
- KwdEn :
- Alzheimer Disease (complications), Alzheimer Disease (pathology), Alzheimer Disease (physiopathology), Apraxia, Apraxias (complications), Apraxias (pathology), Apraxias (physiopathology), Basal Ganglia (physiopathology), Degeneration, Gait Apraxia (physiopathology), Humans, Huntington Disease (complications), Huntington Disease (physiopathology), Huntington disease, Lewy Bodies (pathology), Magnetic Resonance Imaging, Movement Disorders (complications), Movement Disorders (pathology), Movement Disorders (physiopathology), Nervous system diseases, Neuropsychological Tests, Parkinson Disease (complications), Parkinson Disease (pathology), Parkinson Disease (physiopathology), Parkinson disease, Psychomotor Performance.
- MESH :
- complications : Alzheimer Disease, Apraxias, Huntington Disease, Movement Disorders, Parkinson Disease.
- pathology : Alzheimer Disease, Apraxias, Lewy Bodies, Movement Disorders, Parkinson Disease.
- physiopathology : Alzheimer Disease, Apraxias, Basal Ganglia, Gait Apraxia, Huntington Disease, Movement Disorders, Parkinson Disease.
- Humans, Magnetic Resonance Imaging, Neuropsychological Tests, Psychomotor Performance.
Abstract
The definition of apraxia specifies that the disturbance of performed skilled movements cannot be explained by the more elemental motor disorders typical of patients with movement disorders. Generally this does not present a significant diagnostic problem when dealing with ‘higher-level’ praxic disturbances (e.g. ideational apraxia), but it can be a major confound in establishing the presence of limb-kinetic apraxia. Most motor disturbances characteristic of extrapyramidal disorders, particularly bradykinesia and dystonia, will compromise the ability to establish the presence of loss of dexterity and deftness that constitutes this subtype. The term ‘apraxia’ has also been applied to other motor disturbances, such as ‘gait apraxia’ and ‘apraxia of eyelid opening’, that perhaps are misnomers, demonstrating the lack of a coherent nomenclature in this field. Apraxia is a hallmark of corticobasal degeneration (CBD) and historically this has received the most attention among the movement disorders. Corticobasal degeneration is characterized by various forms of apraxia affecting limb function, particularly ideomotor apraxia and limb-kinetic apraxia, although buccofacial and oculomotor apraxia can be present as well. The syndrome of parkinsonism and prominent apraxia, designated the ‘corticobasal syndrome’ (CBS), may be caused by a variety of other central nervous system pathologies including progressive supranuclear palsy (PSP), Alzheimer's disease, dementia with Lewy bodies and frontotemporal dementias. Distinct from the CBS, PSP and Parkinson's disease can demonstrate varying degrees of apraxia on selected tests, especially in those patients with more severe cognitive dysfunction. Diseases that cause the combination of apraxia and a primary movement disorder most often involve a variety of cerebral cortical sites as well as basal ganglia structures. Clinical-pathological correlates and functional imaging studies are compromised by both this diffuse involvement and the confusion experienced in the clinical evaluation of apraxia in the face of the additional elemental movement disorders. Finally, although apraxia results in clear disability in patients with the CBS, it is not clear how milder ideomotor apraxia found on specific testing contributes to patients' overall day-to-day motor disability.
Url:
DOI: 10.1093/brain/awh560
Affiliations:
Links toward previous steps (curation, corpus...)
- to stream Istex, to step Corpus: 002311
- to stream Istex, to step Curation: 002311
- to stream Istex, to step Checkpoint: 000C41
- to stream PubMed, to step Corpus: 001251
- to stream PubMed, to step Curation: 001251
- to stream PubMed, to step Checkpoint: 001251
- to stream Ncbi, to step Merge: 000545
- to stream Ncbi, to step Curation: 000545
- to stream Ncbi, to step Checkpoint: 000545
- to stream Main, to step Merge: 002D45
- to stream PascalFrancis, to step Corpus: 000899
- to stream PascalFrancis, to step Curation: 000424
- to stream PascalFrancis, to step Checkpoint: 000820
- to stream Main, to step Merge: 002E88
- to stream Main, to step Curation: 002A13
Le document en format XML
<record><TEI wicri:istexFullTextTei="no"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Apraxia in movement disorders</title>
<author><name sortKey="Zadikoff, Cindy" sort="Zadikoff, Cindy" uniqKey="Zadikoff C" first="Cindy" last="Zadikoff">Cindy Zadikoff</name>
</author>
<author><name sortKey="Lang, Anthony E" sort="Lang, Anthony E" uniqKey="Lang A" first="Anthony E." last="Lang">Anthony E. Lang</name>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">ISTEX</idno>
<idno type="RBID">ISTEX:D1728C98B247012F10A6A47FD90115BDF487FF5B</idno>
<date when="2005" year="2005">2005</date>
<idno type="doi">10.1093/brain/awh560</idno>
<idno type="url">https://api-v5.istex.fr/document/D1728C98B247012F10A6A47FD90115BDF487FF5B/fulltext/pdf</idno>
<idno type="wicri:Area/Istex/Corpus">002311</idno>
<idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Corpus" wicri:corpus="ISTEX">002311</idno>
<idno type="wicri:Area/Istex/Curation">002311</idno>
<idno type="wicri:Area/Istex/Checkpoint">000C41</idno>
<idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Checkpoint">000C41</idno>
<idno type="wicri:doubleKey">0006-8950:2005:Zadikoff C:apraxia:in:movement</idno>
<idno type="wicri:source">PubMed</idno>
<idno type="RBID">pubmed:15930045</idno>
<idno type="wicri:Area/PubMed/Corpus">001251</idno>
<idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Corpus" wicri:corpus="PubMed">001251</idno>
<idno type="wicri:Area/PubMed/Curation">001251</idno>
<idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Curation">001251</idno>
<idno type="wicri:Area/PubMed/Checkpoint">001251</idno>
<idno type="wicri:explorRef" wicri:stream="Checkpoint" wicri:step="PubMed">001251</idno>
<idno type="wicri:Area/Ncbi/Merge">000545</idno>
<idno type="wicri:Area/Ncbi/Curation">000545</idno>
<idno type="wicri:Area/Ncbi/Checkpoint">000545</idno>
<idno type="wicri:Area/Main/Merge">002D45</idno>
<idno type="wicri:source">INIST</idno>
<idno type="RBID">Pascal:05-0329446</idno>
<idno type="wicri:Area/PascalFrancis/Corpus">000899</idno>
<idno type="wicri:Area/PascalFrancis/Curation">000424</idno>
<idno type="wicri:Area/PascalFrancis/Checkpoint">000820</idno>
<idno type="wicri:explorRef" wicri:stream="PascalFrancis" wicri:step="Checkpoint">000820</idno>
<idno type="wicri:doubleKey">0006-8950:2005:Zadikoff C:apraxia:in:movement</idno>
<idno type="wicri:Area/Main/Merge">002E88</idno>
<idno type="wicri:Area/Main/Curation">002A13</idno>
<idno type="wicri:Area/Main/Exploration">002A13</idno>
</publicationStmt>
<sourceDesc><biblStruct><analytic><author><name sortKey="Zadikoff, Cindy" sort="Zadikoff, Cindy" uniqKey="Zadikoff C" first="Cindy" last="Zadikoff">Cindy Zadikoff</name>
<affiliation wicri:level="4"><country xml:lang="fr">Canada</country>
<wicri:regionArea>Morton and Gloria Shulman Movement Disorders Center, Toronto Western Hospital, University of Toronto, Toronto, Ontario</wicri:regionArea>
<orgName type="university">Université de Toronto</orgName>
<placeName><settlement type="city">Toronto</settlement>
<region type="state">Ontario</region>
</placeName>
</affiliation>
</author>
<author><name sortKey="Lang, Anthony E" sort="Lang, Anthony E" uniqKey="Lang A" first="Anthony E." last="Lang">Anthony E. Lang</name>
<affiliation wicri:level="4"><country xml:lang="fr">Canada</country>
<wicri:regionArea>Morton and Gloria Shulman Movement Disorders Center, Toronto Western Hospital, University of Toronto, Toronto, Ontario</wicri:regionArea>
<orgName type="university">Université de Toronto</orgName>
<placeName><settlement type="city">Toronto</settlement>
<region type="state">Ontario</region>
</placeName>
</affiliation>
</author>
</analytic>
<series><idno type="ISSN">0006-8950</idno>
</series>
</biblStruct>
</sourceDesc>
<seriesStmt><idno type="ISSN">0006-8950</idno>
</seriesStmt>
</fileDesc>
<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Alzheimer Disease (complications)</term>
<term>Alzheimer Disease (pathology)</term>
<term>Alzheimer Disease (physiopathology)</term>
<term>Apraxia</term>
<term>Apraxias (complications)</term>
<term>Apraxias (pathology)</term>
<term>Apraxias (physiopathology)</term>
<term>Basal Ganglia (physiopathology)</term>
<term>Degeneration</term>
<term>Gait Apraxia (physiopathology)</term>
<term>Humans</term>
<term>Huntington Disease (complications)</term>
<term>Huntington Disease (physiopathology)</term>
<term>Huntington disease</term>
<term>Lewy Bodies (pathology)</term>
<term>Magnetic Resonance Imaging</term>
<term>Movement Disorders (complications)</term>
<term>Movement Disorders (pathology)</term>
<term>Movement Disorders (physiopathology)</term>
<term>Nervous system diseases</term>
<term>Neuropsychological Tests</term>
<term>Parkinson Disease (complications)</term>
<term>Parkinson Disease (pathology)</term>
<term>Parkinson Disease (physiopathology)</term>
<term>Parkinson disease</term>
<term>Psychomotor Performance</term>
</keywords>
<keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Alzheimer Disease</term>
<term>Apraxias</term>
<term>Huntington Disease</term>
<term>Movement Disorders</term>
<term>Parkinson Disease</term>
</keywords>
<keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Alzheimer Disease</term>
<term>Apraxias</term>
<term>Lewy Bodies</term>
<term>Movement Disorders</term>
<term>Parkinson Disease</term>
</keywords>
<keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Alzheimer Disease</term>
<term>Apraxias</term>
<term>Basal Ganglia</term>
<term>Gait Apraxia</term>
<term>Huntington Disease</term>
<term>Movement Disorders</term>
<term>Parkinson Disease</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Humans</term>
<term>Magnetic Resonance Imaging</term>
<term>Neuropsychological Tests</term>
<term>Psychomotor Performance</term>
</keywords>
<keywords scheme="Pascal" xml:lang="fr"><term>Apraxie</term>
<term>Chorée Huntington</term>
<term>Dégénérescence</term>
<term>Parkinson maladie</term>
<term>Système nerveux pathologie</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">The definition of apraxia specifies that the disturbance of performed skilled movements cannot be explained by the more elemental motor disorders typical of patients with movement disorders. Generally this does not present a significant diagnostic problem when dealing with ‘higher-level’ praxic disturbances (e.g. ideational apraxia), but it can be a major confound in establishing the presence of limb-kinetic apraxia. Most motor disturbances characteristic of extrapyramidal disorders, particularly bradykinesia and dystonia, will compromise the ability to establish the presence of loss of dexterity and deftness that constitutes this subtype. The term ‘apraxia’ has also been applied to other motor disturbances, such as ‘gait apraxia’ and ‘apraxia of eyelid opening’, that perhaps are misnomers, demonstrating the lack of a coherent nomenclature in this field. Apraxia is a hallmark of corticobasal degeneration (CBD) and historically this has received the most attention among the movement disorders. Corticobasal degeneration is characterized by various forms of apraxia affecting limb function, particularly ideomotor apraxia and limb-kinetic apraxia, although buccofacial and oculomotor apraxia can be present as well. The syndrome of parkinsonism and prominent apraxia, designated the ‘corticobasal syndrome’ (CBS), may be caused by a variety of other central nervous system pathologies including progressive supranuclear palsy (PSP), Alzheimer's disease, dementia with Lewy bodies and frontotemporal dementias. Distinct from the CBS, PSP and Parkinson's disease can demonstrate varying degrees of apraxia on selected tests, especially in those patients with more severe cognitive dysfunction. Diseases that cause the combination of apraxia and a primary movement disorder most often involve a variety of cerebral cortical sites as well as basal ganglia structures. Clinical-pathological correlates and functional imaging studies are compromised by both this diffuse involvement and the confusion experienced in the clinical evaluation of apraxia in the face of the additional elemental movement disorders. Finally, although apraxia results in clear disability in patients with the CBS, it is not clear how milder ideomotor apraxia found on specific testing contributes to patients' overall day-to-day motor disability.</div>
</front>
</TEI>
<affiliations><list><country><li>Canada</li>
</country>
<region><li>Ontario</li>
</region>
<settlement><li>Toronto</li>
</settlement>
<orgName><li>Université de Toronto</li>
</orgName>
</list>
<tree><country name="Canada"><region name="Ontario"><name sortKey="Zadikoff, Cindy" sort="Zadikoff, Cindy" uniqKey="Zadikoff C" first="Cindy" last="Zadikoff">Cindy Zadikoff</name>
</region>
<name sortKey="Lang, Anthony E" sort="Lang, Anthony E" uniqKey="Lang A" first="Anthony E." last="Lang">Anthony E. Lang</name>
</country>
</tree>
</affiliations>
</record>
Pour manipuler ce document sous Unix (Dilib)
EXPLOR_STEP=$WICRI_ROOT/Wicri/Canada/explor/ParkinsonCanadaV1/Data/Main/Exploration
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 002A13 | SxmlIndent | more
Ou
HfdSelect -h $EXPLOR_AREA/Data/Main/Exploration/biblio.hfd -nk 002A13 | SxmlIndent | more
Pour mettre un lien sur cette page dans le réseau Wicri
{{Explor lien |wiki= Wicri/Canada |area= ParkinsonCanadaV1 |flux= Main |étape= Exploration |type= RBID |clé= ISTEX:D1728C98B247012F10A6A47FD90115BDF487FF5B |texte= Apraxia in movement disorders }}
This area was generated with Dilib version V0.6.29. |